Academic Research

Professor Ching-Hsiu Tsai and his team investigated four potential phosphorylation sites (S15, S18, T58, and S247) in the movement protein TGBp1 of Bamboo mosaic virus (BaMV). Their study revealed the functional importance of these phosphorylation sites in viral regulation. In particular, phosphorylation-mimicking mutations at S15, S18, and T58 reduced silencing suppressor activity, viral accumulation, and cell-to-cell movement, highlighting the critical role of TGBp1 phosphorylation in modulating BaMV infection. The study was published in Journal of General Virology (Wu et al., 2024).

Article source: https://www.microbiologyresearch.org/content/journal/jgv/10.1099/jgv.0.001945